A new single-cell profiling technique has mapped pre-malignant gene mutations and their effects in solid tissues for the ...

A new LUMC study has changed our understanding of how cells work. Researchers have discovered that the CFAP20 protein acts as ...

Researchers are targeting dormant tumour cells that might explain why some cancers reappear long after successful treatment.

A new study explains how pancreatic tumors use a sugar coating to hide from the immune system and shows that a newly developed antibody can restore immune responses in mice. Pancreatic cancer remains ...

Researchers at The Institute of Cancer Research, London identified the CIP2A–TOPBP1 complex as a master regulator of DNA repair during mitosis, coordinating backup pathways that protect chromosomes ...

A hidden clue may explain why some mutated cells become cancerous and others don’t: how fast they divide. A new study from researchers at Sinai Health in Toronto reveals that the total time it takes ...

Left: Normal cell division with the chromosomes (blue) lined up and ready to be pulled into two separate daughter cells by the two centrosomes (green). Right: In faulty cell division, too many ...

Long before a liver tumor appears, a high-fat diet can push liver cells into a risky survival mode. That is the central ...

A research team at the Medical University of Vienna has discovered a new approach to treating a particularly frequent and ...

From the deep sea to the shallow seafloor, researchers are uncovering unusual sugars that do something extraordinary to ...

The ability of mutations to cause cancer depends on how fast they force cells to divide, Sinai Health researchers have found. The study, led by Dr. Rod Bremner, a Senior Investigator at the ...